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Through clinical observation and data from animal experiments, we suspected that elevated levels of CRH in the brain would have suppressed non-REM sleep or increased waking time.6, 9, 10 In contrast, after SD the orally given CRHR1 antagonist increased non-REM sleep in both heterozygous and homozygous CRH-COE-Nes mice, which implies that under stressful conditions, such as SD, the brain CRH is als
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