"TNF), as well as endotoxins and proinflammatory stimuli such as polyinosine-polycytidine (poly(IC)), lipopolysaccharides (LPS), peptidoglycan (PGN), flagellin, or lipid A activate MAP4K2 by promoting its autophosphorylation.SitesFeature keyPosition(s)LengthDescriptionGraphical viewFeature identifierActions

Describes the interaction between a single amino acid and another chemical entity." . . . .