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  • From a teleological standpoint, one may speculate that the turnover of Th17 cell population is tightly controlled in vivo by mechanisms such as apoptosis and/or AICD pathways [47],[48], IDO-mediated tryptophan-deprivation [49], or galectin-1 signaling [50], mechanisms that might not pertain to the turnover of Tregs [51].
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