The oxalate deficiency of mutant A336 therefore could contribute to its lack of virulence.Mutant A336 is also affected in the production of extracellular enzymes; some of these enzymes, such as phosphatases and the endopolygalacturonase BcPG1, are produced in larger amounts than in the wild-type strain, while others, such as the acid protease ACP1, are not produced at all.