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  • inflammatory mediators such as C-reactive protein, tumor necrosis factor-alpha, and interleukin-6 may contribute to the myocardial oxygen demand-supply mismatch.65 In addition, it has been proposed that myocardial depressive factors (released in the setting of sepsis and other inflammatory states) cause degradation of free troponin, in situ, to lower molecular weight fragments.67 With increased me
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