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  • ather that regulatory, and contribute to the development of SLE. There remains still a lack of understanding as to what causes sema3A and NP-1 to be altered in SLE. The milieu of increased pro-inflammatory cytokines such as IFN-?? and IL-6 may be a factor.Further, sema3A has been reported to be important in the induction of the apoptosis of many immune cells, such as monocytes and macrophages, whe
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