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  • Anti-IL-13 was highly effective in reducing inflammatory pathology while neutralizing IL-17 had little effect (Figure 4E), suggesting that IL-13 is a key pathogenic cytokine in the intestinal inflammation induced by chronic TL1A expression.The TNF receptor DR3 is thought to mediate most, but not all, of the biological effects of TL1A (4, 6, 19).
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