| http://www.w3.org/ns/prov#value | - The ability of classical NSAIDs to inhibit prostaglandin synthesis by cyclooxygenase (COX) isozymes, particularly COX-1, is implicated in these acute exacerbations [3], but it is not known how they activate mast cells, eosinophils, macrophages, epithelium, and other cells to release a range of inflammatory mediators in susceptible subjects.
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