| http://www.w3.org/ns/prov#value | - This increase is a result of a significant loss of GABAergic interneurons, reduced surface expression of GABAA receptors and inhibitory synaptic transmission, and, increases in the surface expression and function of ??7-nAChRs, which subsequently increase excitability within principal neurons in the BLA. With the high incidence of anxiety disorders being reported after TBI [47], it is essential to
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