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  • Thus, understanding the pathophysiology of endothelial dysfunction beyond NO, and in particular with respect to EDHF in these disease states, could be crucial in understanding both the pathophysiology of atherosclerosis and developing novel therapies.AcknowledgmentsProfessor A. Quyyumi was supported by National Institutes of Health Research Grant RO1 HL79115, and in part by PHS Grant UL1 RR025008
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  • nih.gov