| http://www.w3.org/ns/prov#value | - Although typical NF-kB activation is transient, under specific physiological or pathological conditions, inducers such as TNF-a and IL-1 can cause a persistent activation of NF-kB.[17],[18] Activation of NF-kB in the brain is mainly related to the neuronal function[16],[19] and inflammatory response[20] through induction of expression of proinflammatory genes such as TNF-a, IL-1a, IL-6, cyclooxyge
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