| http://www.w3.org/ns/prov#value | - Although some physiological roles of proinflammatory cytokines at the maternal-fetal interface have been described with regard to growth of the placenta and decidua [9], much of the literature supports the concept that excessive or aberrant production of proinflammatory cytokines such as Interleukin (IL)-1??, Tumor necrosis factor (TNF)-??, and Interferon (IFN)-?? at the maternal-fetal interface i
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