| http://www.w3.org/ns/prov#value | - Furthermore, cilostazol attenuated the TNFalpha-induced gene expression of various pro-inflammatory and cell adhesion molecules, such as vascular cell adhesion molecule-1, E-selectin, intercellular adhesion molecule-1, monocyte chemoattractant protein-1 (MCP-1), and PECAM-1 in HUVEC. RNA interference of AMPKalpha1 or the AMPK inhibitor compound C attenuated cilostazol-induced inhibition of NF-kapp
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