| http://www.w3.org/ns/prov#value | - Mutated, it is involved in up to 70% of human tumors, being responsible of cell growth arrest, senescence, apoptosis in response to an array of stimuli such as DNA damages (DSB, or double-strand-breaks), hypoxia, telomeres shortening, cell adhesion, oncogene activation and other molecular and cellular stresses [15].
|
