| http://www.w3.org/ns/prov#value | - Recent advances suggest that cholinergic control of keratinocytic adhesion and proinflammatory cytokines, particularly TNF-??, is involved in the pathogenesis of PV. Nguyen et al8 report that nondesmoglein autoantibodies to cholinergic receptors can also induce clinical features of PV. These cholinergic receptors regulate adhesion and motility of keratinocytes, and their activation by autoantibodi
|
