| http://www.w3.org/ns/prov#value | - A recent study by Peters et al. [31], found that 87% of individuals homozygous for the UGT1A1*28 allele, were also homozygous for the UGT1A6*2 allele, implying that such individuals might have reduced capacity to glucuronidate bilirubin, as well as several drugs such as aspirin and coumarin due to the combined UGT1A1*28 and UGT1A6*2 genotypes.
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