| http://www.w3.org/ns/prov#value | - Mechanistically, our data suggest that these defects are in part due to decreased phosphorylation of Src and p190RhoGAP (GTPase-activating protein), yielding increased Rho/Rho kinase (ROCK) signaling and inefficient recruitment of the p95 paxillin kinase linker???PAK-interacting exchange factor???p21-activated kinase (PKL???PIX???PAK) complex to
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