| http://www.w3.org/ns/prov#value | - Based on our current understanding, we propose three, not necessarily mutually exclusive, mechanisms by which sterile endogenous stimuli trigger inflammation: activation of PRRs by mechanisms similar to those used by microorganisms and PAMPs; release of intracellular cytokines and chemokines, such as IL-1??, that activate common pathways downstream of PRRs; and direct activation by receptors that
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