| http://www.w3.org/ns/prov#value | - hio, USA.AbstractOverexpression of GALGT2 in skeletal muscle can stimulate the glycosylation of ?? dystroglycan and the upregulation of normally synaptic dystroglycan-binding proteins, some of which are dystrophin and laminin ??2 surrogates known to be therapeutic for several forms of muscular dystrophy.
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