| http://www.w3.org/ns/prov#value | - e from these experiments that activation of the MKK3/6-p38 pathway is necessary and sufficient to induce the cytoplasmic accumulation of hnRNP A1 in response to cellular stress.Changes in Alternative Splicing Regulation after Activation of the MKK3/6-p38 PathwayAlternative splicing is a widespread mechanism for the spatial or temporal control of gene expression. hnRNP A1 and other closely related
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